Showing posts with label ensure. Show all posts
Showing posts with label ensure. Show all posts

Tuesday, 30 July 2013

Protein moulds RNA to ensure that activating factors can hold on to it

Main Category: Genetics
Article Date: 30 Jul 2013 - 0:00 PDT Current ratings for:
Protein moulds RNA to ensure that activating factors can hold on to it
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X chromosomes are very special genetic material. They differ in number between men and women. To achieve equality between sexes, one out of two X chromosomes in women is silenced. In flies, the opposite happens: in male flies, the only available X chromosome is highly activated, to compensate for the absence of the second X-chromosome. Researchers from the Max Planck Institute of Immunobiology and Epigenetics (MPI-IE) in Freiburg have now shown how the RNA molecules and proteins involved in the activation find and stick to each other. Similar to a monkey that grabs a liana with hands and feet, one of the proteins holds on to the RNA. Then it moulds the molecular liana with its hands and thus generates a dynamic RNA - protein meeting place.

Just a few years ago, they were assumed to be genetic trash: DNA sequences that are not translated into proteins. But this has rapidly changed during the last years. Nowadays, it is widely known among scientists that much of the DNA is transcribed into RNA that, in turn, can act as gene regulator and structural element. Also in the regulation of sex chromosomes, RNA plays a central role. In both female humans and male flies one X chromosome is covered by a protein-RNA complex. In humans, this leads to chromosome silencing, while in flies it results in a double activation of the chromosome. Misregulation is lethal. Although known for many years, the interaction between the central proteins and the distinct role of the RNA strand was unclear.

Asifa Akhtar of the MPI-IE and her team now unravelled the function of the RNA and the interaction of the proteins. The protein MLE that is known to be a central player in X chromosome activation binds to the RNA in a very special manner. Like a monkey that grabs a liana with hands and feet, the protein grabs the RNA in two different ways. While one site is a simple anchor (the feet), the other (the hands) changes the form of the RNA. "The protein MLE moulds the RNA strand. This allows MLE to bind the RNA in a dynamic manner", says Asifa Akhtar, head of the study. Like one monkey helping the other to catch the liana MLE could thus help other proteins to grab the RNA strand. Thus, the whole X chromosome can be covered by the RNA-protein complex.

During his PhD work, first author Ibrahim Ilik investigated why MLE was found at the same places on the X chromosome but did not directly interact with other proteins. "The biochemical and the biological results seemed to point in different directions in the beginning", says Ilik. "But when we realised that the proteins bind highly specifically to certain regions of the very long RNA, this was a very exciting moment."

The researchers also found that individual mutations in the RNA hardly harm the protein-RNA binding. Only multiple mutations lead to a non-functional RNA and thus to lethality of male flies. "The system is very robust for evolutionary influences. This shows how important it is for the survival of the animals. In this, RNA could provide the necessary plasticity", says Akhtar. The scientists now want to explore the evolutionary conservation of the RNA-protein system and its equivalent in mammals.

Scientists at the Max Planck Institute of Immunobiology and Epigenetics (MPI-IE) in Freiburg investigate the development of the immune system over the course of evolution and during lifetime. They analyse genes and molecules that are important for immune cells maturation and activation. Researchers in the field of epigenetics investigate the inheritance of traits that are not caused by changes in the DNA sequence. Epigenetic research is expected to lead to a better understanding of many complex diseases, such as cancer and metabolic disorders.

Article adapted by Medical News Today from original press release. Click 'references' tab above for source.
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Friday, 26 July 2013

To ensure proper cell division, centrioles must be kept in check

Main Category: Genetics
Also Included In: Cancer / Oncology;  Biology / Biochemistry
Article Date: 25 Jul 2013 - 0:00 PDT Current ratings for:
To ensure proper cell division, centrioles must be kept in check
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The duplication of cellular contents and their distribution to two daughter cells during cell division are amongst the most fundamental features of all life on earth. How cell division occurs and is coordinated with organismal development is a subject of intense research interest, as is how this process malfunctions in the development of tumors. Alex Dammermann and his team from the Max F. Perutz Laboratories (MFPL) of the University of Vienna and the Medical University of Vienna, together with his collaborators from the Institute of Molecular Pathology (IMP), have been investigating how the duplication of one key component of the cell division machinery, named centrioles, is coordinated with the cell cycle - the series of events that lead to a cell's division. Their results are published in the journal Current Biology.

Centrioles - orchestrators of cell division

When our cells divide, their genetic material - in the form of X-shaped chromosomes - is aligned in the middle of the cell and segregated to opposite poles of the cell by a spindle of long tubular fibers, so-called microtubules. The structures that organize the two poles of the spindle in animal cells are called centrosomes. Each centrosome consists of two cylindrically shaped centrioles that are positioned perpendicular to each other and surrounded by an amorphous dense mass called the pericentriolar material (PCM). At the end of cell division, the two centrioles inherited by each daughter cell separate, and later each of them forms a new centriole. This ensures that another bipolar spindle can be set up by two centrosomes when the cell divides again. Precise control of centriole separation and duplication is therefore essential for successful cell division. Abnormal centrosome numbers are commonly observed in human cancers and are thought to be at least in part responsible for the improper distribution of the genetic material that is a hallmark of many cancer cells.

The PCM -the glue that keeps centrioles together

Until now, it was unclear how centrioles are held together and how their separation at the end of cell division is so precisely regulated. Gabriela Cabral, a PhD student in the lab of Alex Dammermann at the Center for Molecular Biology of the University of Vienna, explains: "Many people thought that centrioles are held together by the same glue as chromosomes, a substance called cohesin, which is destroyed during cell division. We found this to be true only in the very specialized circumstances surrounding fertilization. In all other cases, as in the subsequent cell divisions following fertilization, the glue that holds centrioles together is actually the PCM." These findings explain previously conflicting data on the mechanism of centriole separation. Alex Dammermann adds: "The surprising finding that there are actually two cellular mechanisms for controlling centriole separation was only possible because we use the nematode worm C. elegans as our model organism. Would we have used cell cultures we would have never found that centriole separation works differently in different developmental contexts".

Stem cell fate and cancer

The dense mass of the PCM that entraps the sister centrioles is itself disassembled at the end of cell division. The microtubules that are responsible for separating the genetic material also appear to be involved in pulling the PCM and centrioles apart. This tightly regulated process is critical to ensure that both daughter cells will later have the correct centrosome numbers when they divide. This is important to avoid missegregation of the genetic material, which may result in cell death or tumor formation. Interestingly, centrosomes have also been linked to the segregation of cell fate determinants. Gabriela Cabral explains: "When a stem cell divides, it doesn't produce two identical daughter cells as normal cells do. It produces another stem cell and a daughter cell that may differentiate into one of many specialized cell types." What these cell fate determinants are and how they are distributed when a stem cell divides is another big question. However, it is known that centrosomes are also involved in this process. Alex Dammermann says: "Our results show that the PCM still harbors many surprises. One of our current research goals is to examine how this largely mysterious accumulation of cellular material is organized and we hope that a better knowledge of this will help us understand how centrosomes perform their manifold functions in the cell."

Article adapted by Medical News Today from original press release. Click 'references' tab above for source.
Visit our genetics section for the latest news on this subject.

Original publication in Current Biology: Gabriela Cabral, Sabina Sanegre Sans, Carrie R. Cowan, and Alexander Dammermann: Multiple mechanisms contribute to centriole separation in C. elegans. Current Biology (July 2013). DOI: http://dx.doi.org/10.1016/j.cub.2013.06.043

University of Vienna

Please use one of the following formats to cite this article in your essay, paper or report:

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University of Vienna. "To ensure proper cell division, centrioles must be kept in check." Medical News Today. MediLexicon, Intl., 25 Jul. 2013. Web.
26 Jul. 2013. APA

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'To ensure proper cell division, centrioles must be kept in check'

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View the original article here